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Inhibition of macrophage fatty acid -oxidation exacerbates palmitate-induced inflammatory and endoplasmic reticulum stress responses

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This work was supported by the Italian Ministry for Education, University and Research PRIN grant 2017483NH8 to R.P

Highly reactive species such as hydroxyl radical (OH) and perhydroxyl radical (HO 2 ) emerge from secondary interactions involving O 2 - NAFLD progression is further driven by insulin resistance (IR), ER stress, and the activation of the unfolded protein response (UPR) pathway [52, 53]